![]() Involvement can invovle single or mutiple muscle groups.Ĭan be caused by any process that damages the motor unit. Speech characteristics / Non-speech exam findings: Cranial & spinal respiratory nerves - Intro ![]() May manifest in any or all respiratory, phonatory, or articulatory speech components.ĭue to muscle weakness and reduced muscle tone, affects speed, range, and accuracy of speech. Reflect problems in the nuclei, neurons, or neuromuscular junctions that make up the motor units of the fnal common pathway. Produced by injury of malfunction of one or more of the cranial or spinal nerves. ![]() Strained-harsh voice quality, slow speech rate, and slow/regular AMRs are the most distinctive clues to the presence of spastic dysarthria Salient characteristics of spastic dysarthria 2Ĭhief disturbances are slowness and reduced range of individual and repetitive movements, reduced force of movement, and excessive or biased muscle tone or spasticity. Intelligibility is often affected due to the involvement of multiple speech valves. Salient characteristics of spastic dysarthria 1Ĭonversational speech, reading, & speech AMR are the most useful tasks for eliciting salient chracteristics.Īssociated with impaired movement patterns, rather than weakness of individual muscles.ĭeficits seen at all speech valves and for all components of speech, though not always equally. How easily the response is triggered if often related to the emotional loading of the interaction. May fluctuate between laughing and crying or have difficulty inhibiting laughter or crying once they begin. Patient may cry or laugh in a stereotypic manner for no apparent reason. Spastic Dysarthria: non-oral speech exam 2ĭisplay of pseudobulbar affect (lability of affect) is common. The face may be held in a somewhat fixed, subtle smiling or pouting posture. Spastic Dysarthria: non-oral speech exam 1ĭrooling is common, likely due to decreased frequency of swallowing. Predominant causes: Degenerative (e.g., ALS), vascular, and traumatic disorders. ![]() Produced by bilateral damage to the direct and indirect activation pathways of the Central Nervous System (CNS), often associaled with upper motor neuron disease.Ĭharacteristics reflect the combined effects of weakness and spasticity (excessive muscle tone) in a way that slows movement and reduces its range and force. Sometimes perceived as slurred speech with a "drunken quality". Halting, imprecise, jerky, poorly coordinated, and lacking in speed and fluidity. Produces slowness and inaccuracy in force, range, timing, & direction of speech movement. Associated with damage to the cerebellar control circuit,Ĭausing in-coordination and poor muscle tone. ![]()
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